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Consequently, the further inhibition of Gln metabolism and fatty acid metabolism through the suppression of ASCT2 expression, which poses a significant threat to the energy supply required for cancer cell proliferation, emerges as a promising anticancer target

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Deutsch Foundation, the National Science Foundation (CBET-1435957), the Department of Defense and Defense Threat Reduction Agency (HDTRA1-13-1-0037)

Ferrannini & DeFronzo, 2015)