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Taken together, the Bcl-2/Bax shift, the caspase-3 reduction, and the PARP reduction tell a consistent story: AHK-Cu appears to push dermal papilla cells away from self-destruction and toward survival

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Different mechanisms, overlapping tissue endpoints hence the frequent combination in research protocols

Researchers have linked those effects to upregulation of growth factor receptors, including VEGFR2, and to nitric oxide signaling